Balloon Dilation of Atheroma
Pathophysiological Changes and Clinical Implications
Mechanical Plaque Alteration
Compression and Fracture
Balloon inflation compresses the atheroma against the arterial wall, fracturing the plaque and expanding the lumen.
Endothelial Injury
Procedure damages endothelial layer, exposing subendothelial collagen and plaque components, activating platelets.
Acute Vascular Responses
Elastic Recoil
Artery may partially collapse post-deflation, especially in calcified plaques, reducing lumen gain.
Dissection & Vasospasm
Overstretching causes microtears; endothelial dysfunction impairs nitric oxide release.
Thrombotic & Inflammatory
Thrombosis Formation
Exposed components trigger platelet adhesion. Antiplatelet therapy critical for prevention.
Inflammatory Response
Neutrophils and macrophages infiltrate, releasing cytokines and growth factors driving SMC activation.
Healing & Remodeling
Neointimal Hyperplasia
Migrating SMCs proliferate in intima, producing extracellular matrix. Key contributor to restenosis.
Vascular Remodeling
Negative: vessel contraction reduces lumen. Positive: external membrane expansion preserves patency.
Long-Term Outcomes
Restenosis Risk
Historically, 30–50% experienced renarrowing. Drug-eluting stents reduce this by releasing antiproliferative agents like sirolimus.
Disease Progression
Balloon dilation doesn't halt underlying atherosclerosis. Comprehensive medical management remains essential for long-term outcomes.
Clinical Summary
Balloon dilation induces acute mechanical benefits but triggers complex healing responses. Understanding these mechanisms underscores the importance of adjunctive therapies and advanced technologies to optimize patient outcomes.